The Relation Between Passive Smoking and Diabetes by Smith Baker

The Relation Between Passive Smoking and Diabetes.

Over the past decade, the growing incidence of lung cancer has closely been associated to active and passive smoking. However, the health risks associated with second-hand smoke are not just limited to lung diseases. It has been proven now, that passive smoking is closely linked to increased risks of diabetes. This risk has been proven after a careful analysis of the impact of passive tobacco smoke on glucose intolerance. Individuals who inhale second hand smoke are more likely to become glucose intolerant.
Statistics reveal that the total number of diabetic or glucose intolerant individuals is rising. This alarming trend is owed to increased urbanization, obesity and sedentary lifestyles. It has been predicted by The International Diabetes Federation that the total number of people with diabetes or glucose intolerance is said to increase to 380 million in the year 2025. This prediction suggests that diabetes has emerged as a major health priority for individuals across the globe. Currently, smoking accounts for almost six million premature deaths worldwide. The projected number is expected to increase in the current years. It would be safe to assume that smoking is indeed one of the leading causes of (avoidable) deaths worldwide. And now evidence accumulated from a host of studies has developed major links between these two factors.
Research suggests that passive smokers, despite having limited exposure to smoke as compared to that of smokers, are at a higher risk for Diabetes, given the fact that it contains a surprisingly higher concentration of certain toxins as compared to first hand smoke. The impact of these concentrated toxins on the pancreas is detrimental, resulting in a reduction in the beta cell functionality.
In order to understand the association between diabetes type 2 and passive smoking, there is a need for closer and much detailed analysis. There is little doubt the fact that active and passive smoking both have been linked to higher risks of diabetes amongst individuals. The prime reason behind this is that tobacco smoke results in a number of changes in the systemic functionality of the body; particularly by increasing the oxidative stress in the body. Free radicals in cigarette smoke also result in pancreatic inflammation, vascular issues and induce endothelial dysfunction. Each of the affects has long term impact on the total insulin resistance of the body, consequently resulting in diabetes. It is worth mentioning here that despite the fact that the observed BMI of smokers is less than that as compared to nonsmokers, there lies a difference in the fat distribution profile. Smokers tend to have a greater metabolically adverse profile which is highly prone to central adiposity. In addition, smoking also causes a direct impact on the functionality of beta cells. All this evidence suggests the plausible relation between smoking and diabetes.
Currently, the risks associated with smoking are largely underestimated. There is a need for further research on both active and passive smoking in order to ascertain the actual magnitude of the health risks associated with smoking.

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